谢谢雨中鸟，
谢谢绿箩，
谢谢advice
谢谢宝子妈
谢谢洋洋
谢谢尘姥姥
谢谢小鹿。
你们和再上面很多的贴说是我婆婆修来的福。
可我看来，是我修来的福，有这么好的婆婆。我和婆婆一起看韩剧的时候，看到那些韩国婆婆们的嘴脸，我们一起大笑，说我们都不是这样的呀。我永远记得我生斌斌的时候，第一胎呀，我什么都不会，每天中午婆婆就会来帮斌斌洗澡，每天早午晚饭，婆婆总是抱过斌斌，让我安心吃好每一顿饭。
太多太多了。容我以后再说。
昨晚十一点五十分到达医院，我接了外甥女的班，她临走前说，护士刚刚给祖母吊了针，会有很多尿的。果不然，婆婆几乎每半小时就湿了尿布。虽然有助护来换尿布的，但我每次都翻翻看，确定是尿湿了才把助护叫来。要是谎报军情，让助护白走一趟，那多不好意思啊。
夜里三点，我坐了下来，开了电视看。平常我哪有空看电视呢，我突然发现了一个事实，哎，我到来已经三个小时零十分钟，婆婆居然没有叫我给她喝水呢。过去的一个月，婆婆几乎十分钟五分钟要求喝水的。
哇，医生都给婆婆开了什么神丹妙药呢？好奇的我走到输液架那，看见有四个输液袋子。
1.0.15% potassium 5% dextrose 0.45sodium
2.5% dextrose
3.0.9%sodium
4.200ml highly concentrated potassium
看着看着，我发现怎么输液没有流动呢？我把护士叫来，这个护士叫Joe， 是个胡子鬼子佬，他动了动机器，说，好了。他走了以后，我翻开婆婆的被子，我要检查一下婆婆的针口有没有问题，上次住院，婆婆的针口穿破了血管，整只手都变猪手了。好在我有看，天呀，这次没有变猪手，却是看到根本输液管没有接上的，这叫啥输液呀？我忙跑到护士站把Joe 叫了回来。哼，这个护士太马虎了。给他个中文名就叫马虎吧。
记得上次婆婆住院的时候，那个男护士是中国人，却不会说中国话，每天早上来抽血的，是一个说中国话的中国男。这家医院好多男护士噢！
三点半，婆婆说她热，我用我的脸贴着婆婆的脸，我知道她是低烧了。我的这个方法，以前用在我两儿子小时候，很准的。护士站就在病房对面，我去到那，看见Joe 坐着，我往他招手，他没有站起来，也没有走出来，隔得老远的，眼睛瞪大大地看着我，意思是，又怎么了？我不管那护士站里有好几个护士在，我大大声说，我婆婆发烧了。他懒洋洋地走到房间来，也不探热。却问我，你怎么知道她发烧了，我说，我摸到的，而且婆婆说她热。这个死鬼Joe说，oh,no,no,no ,如果发烧，她会觉得冷而不是热。我说，发高烧才会发冷，她现在是低烧，她这么年纪大的人，那怕是一丁点的烧，都要很注意的，请你现在给她探热！Joe 拿了机器来，一探，100.2
明明就是有烧，他却又说：这不算什么烧。
我说：我婆婆现在肺有infection ，连着两天医生给消炎药，下午已经温度正常了，现在又重上100度多，这就是问题，请你记录下来，给医生看，不然，医生以为婆婆已经好了。
Joe叫了一个助护拿了两袋雪，放在婆婆的腋下，婆婆说，舒服多了。到天亮再次探热是99.5度。
早上六点四十五分，小姑到来，我给小姑说了阿姆的热度，让她留意点。我也给小姑说了这个Joe 的离谱事，小姑说，哎呀，这个死鬼佬，昨晚给阿姆刺针头，刺了好久都刺不到位，痛到阿姆哇哇叫。既不细心又没技术。
我们该给医院投诉他吗？吼吼！
又：网上查了查婆婆的输液药，对症下药了，要是再迟到医院来，由着她一直不停地喝水，尿，喝水，尿，后果太严重了。
这些资料解释了为什么婆婆老是要喝水，老是要尿却又好象尿不出。
婆婆肾功能不好，直接导致了减少造血，所以她才会严重贫血。心脏因为贫血，加紧工作，所以心跳加速。
我呀，我算个黄绿医生。嘻嘻。
Symptoms of kidney failure include:
High levels of urea in the blood, which can result in:
Vomiting and/or diarrhea, which may lead to dehydration
Nausea
Weight loss
Nocturnal urination
Less frequent urination
More frequent urination, or in greater amounts than usual, with pale urinetion, or in smaller amounts than usual, with dark coloured urine
Blood in the urine
Pressure, or difficulty urinating
Unusual amounts of urination, usually in large quantities
Healthy kidneys produce the hormone erythropoietin that stimulates the bone marrow to make oxygen-carrying red blood cells. As the kidneys fail, they produce less erythropoietin, resulting in decreased production of red blood cells to replace the natural breakdown of old red blood cells. As a result, the blood carries less hemoglobin, a condition known as anemia. This can result in:
Feeling tired and/or weak
Memory problems
Difficulty concentrating
Dizziness
Low blood pressure
Other symptoms include:
Appetite loss, a bad taste in the mouth
Difficulty sleeping
Darkening of the skin
Excess protein in the blood
Potassium is an essential macromineral in human nutrition; it is the major cation (positive ion) inside animal cells, and it is thus important in maintaining fluid and electrolyte balance in the body.
Epidemiological studies and studies in animals subject to hypertension indicate that diets high in potassium can reduce the risk of hypertension and possibly stroke (by a mechanism independent of blood pressure), and a potassium deficiency combined with an inadequate thiamine intake has produced heart disease in rats.
If potassium were removed from the diet, there would remain a minimum obligatory kidney excretion of about 200 mg per day when the serum declines to 3.0–3.5 mmol/L in about one week,[65] and can never be cut off completely, resulting in hypokalemia and even death.
Hypokalemia can result from one or more of the following medical conditions:
Urinary loss
Certain medications can cause excess potassium loss in the urine. Diuretics, including thiazide diuretics (e.g. hydrochlorothiazide) and loop diuretics (e.g. furosemide) are a common cause of hypokalemia. Other medications such as the antifungal, amphotericin B, or the cancer drug, cisplatin, can also cause long-term hypokalemia.
Severe hypokalemia (<3.0 mEq/L) may require intravenous (IV) supplementation. Typically, a saline solution is used, with 20-40 mEq KCl per liter over 3–4 hours. Giving IV potassium at faster rates (20-25 mEq/hr) may predispose to ventricular tachycardias and requires intensive monitoring. A generally safe rate is 10 mEq/hr. Even in severe hypokalemia, oral supplementation is preferred given its safety profile. Sustained release formulations should be avoided in acute settings.
